A case of sudden sensorineural hearing loss complicated with non-ST-segment elevation myocardial infarction
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摘要: 本文报道1例以突发性感音神经性耳聋为首诊断入院的患者,在治疗过程中出现胸闷不适,后诊断为急性非ST段抬高型心肌梗死。尽管突发性感音神经性听力损失(sudden sensorineural hearing loss,SSNHL)的病因尚不清楚,目前认为可能的机制包括病毒感染、内耳微循环障碍、免疫因素、膜迷路积水等,但越来越多的证据表明其潜在的心血管病理机制。近期相关研究揭示了急性心肌梗死(acute myocardial infarction,AMI)和SSNHL之间的关联,本病例的结果进一步支持了SSNHL发展中潜在的血管病理机制,并强调了临床医生处理SSNHL患者时需要警惕AMI的发生和发展。Abstract: This article reports a case of a patient who was admitted to the hospital with sudden sensorineural hearing loss as the initial diagnosis, and developed chest tightness during the course of treatment, which was later diagnosed as acute non-ST-segment elevation myocardial infarction. Although the etiology of sudden sensorineural hearing loss (SSNHL) remains unclear, current theories suggest possible mechanisms including viral infection, inner ear microcirculation disorders, immune factors, and perilymph fistula. However, increasing evidence suggests its potential cardiovascular pathophysiological mechanisms. Recent studies have revealed the association between acute myocardial infarction (AMI) and SSNHL. The results of this case further support the vascular pathophysiological mechanisms underlying the development of SSNHL and emphasize the need for clinicians to be vigilant about the occurrence and progression of AMI in the management of SSNHL patients.
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Key words:
- Sudden sensorineural hearing loss /
- Myocardial infarction /
- Risk factors
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表 1 咽鼓管功能检查(daPa)
Table 1. Eustachian tube function examination(daPa)
项目 初始峰压力 valsalva动作后压力 再次吞咽后压力 功能 右耳 -10 -102 -102 欠通畅 左耳 -5 -21 -24 欠通畅 表 2 镫骨肌反射
Table 2. Stapedius reflex
项目 类别 0.5 Hz(dB) 1 kHz(dB) 2 kHz(dB) WB(dB) 镫骨肌声反射衰减1 kHz(同侧) 右侧 同侧 未引出 未引出 未引出 未引出 未引出 对侧 未做 未做 未做 未做 左侧 同侧 80 90 80 95 阴性 对侧 未做 未做 未做 未做 -
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